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WHAT YOU NEED TO KNOW Reverse Brain Trauma And Inhibit Neurodegeneration Mainstream therapies have been largely unable to alleviate the progressive symptoms of traumatic brain injury, which often occur years after the original trauma and can include devastating cognitive, emotional, and physical effects. Recognizing that brain trauma triggers sex hormone deficiencies that can produce these worsening effects, some physicians are using an innovative sex hormone replacement technique that uses subcutaneous pellets to deliver bioidentical hormone replacement. Patients report rapid reversal of symptoms. Also, at a time of exploding rates of dementia and Alzheimer’s disease, accumulating evidence suggests that by replenishing diminished sex hormones, aging individuals can help inhibit these devastating brain-wasting diseases. Chronic Traumatic Encephalopathy (CTE) When former heavyweight boxing champion Muhammad Ali was diagnosed with Parkinson’s syndrome in 1984, 10 it was natural to blame his sport. Repeated head blows make professional boxers prone to brain damage, including parkinsonism, tremors, and a severe form of TBI known as chronic traumatic encephalopathy (CTE)—which is the dementia-like disease associated with repeated concussions. 11 CTE develops in about 20% of pro boxers. 12 The repeated concussions suffered in football have been making headlines for some time now. Decades ago, football players knew they were assuming some risk of long-term damage to knees, back, or even the neck—but the risk of devastating cognitive damage was not well-known. Offensive lineman Joe DeLamielleure played his final year in the NFL in 1985 at the age of 34. 13 But years before being named to the Pro Football Hall of Fame in 2003, he began showing TBI symptoms. 14 “You turn 50 and suddenly, things change,” he says. “I thought it was depression, but there were other things besides that. High anxiety. I never slept.” His symptoms included headaches, bursts of anger, and a 68% hearing loss in his left ear, which he attributes to years of right-handed defensive linemen slapping him in the head. “I lived football, I loved football,” DeLamielleure says. “I look at how I am now and I think, ‘Is this a temporary thing or am I going to end up like Mike Webster?’” 15 Webster, a former offensive lineman and member of the Pro Football Hall of Fame, was just 50 when he died, after spending his final years suffering from dementia and parkinsonian symptoms. In his autopsy, CTE was clearly evident. The extensive brain damage included many diffuse amyloid plaques and neurofibrillary tangles, and the small projections from brain neurons known as neuritic threads were also found to contain tau protein. 16 Amyloid and tau are implicated in neurodegenerative diseases, such as Alzheimer’s. 17 CTE effects frequently include memory and decision-making difficulties, mood and behavior problems—including depression and hopelessness—or sudden, violent behavior. Sometimes, there are no symptoms at all. 18 Evidence suggests that a high portion of retired football players develop CTE. 19,20
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